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2015). These reports, taken together, recommend that, Whilst a immediate activation of GPR55 or PPAR‐α takes place, PEA can produce its anti‐inflammatory motion from the intestine also through indirect activation of CB1 and CB2 receptors, probably on account of the power of the compound to potentiate the action of endocannabinoids at these rec
2015). These scientific tests, taken collectively, suggest that, Despite the fact that a immediate activation of GPR55 or PPAR‐α occurs, PEA can produce its anti‐inflammatory action during the intestine also by means of oblique activation of CB1 and CB2 receptors, possibly as a consequence of the ability of this compound to potentiate the moti